Log in to save to my catalogue

EZH2-driven immune evasion defines high-risk pediatric AML with t(16;21) FUS::ERG gene fusion

EZH2-driven immune evasion defines high-risk pediatric AML with t(16;21) FUS::ERG gene fusion

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_biorxiv_primary_2024_05_14_594150

EZH2-driven immune evasion defines high-risk pediatric AML with t(16;21) FUS::ERG gene fusion

About this item

Full title

EZH2-driven immune evasion defines high-risk pediatric AML with t(16;21) FUS::ERG gene fusion

Publisher

United States: Cold Spring Harbor Laboratory

Journal title

bioRxiv, 2024-06

Language

English

Formats

Publication information

Publisher

United States: Cold Spring Harbor Laboratory

Subjects

Subjects and topics

More information

Scope and Contents

Contents

Despite decades of research, acute myeloid leukemia (AML) remains a remarkably lethal malignancy. While pediatric AML (pAML) carries a more favorable prognosis than adult AML, the past 25 years of large clinical trials have produced few improvements in pAML survival. Nowhere is this more evident than in patients carrying a t(16;21)(p11;q22) translo...

Alternative Titles

Full title

EZH2-driven immune evasion defines high-risk pediatric AML with t(16;21) FUS::ERG gene fusion

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_biorxiv_primary_2024_05_14_594150

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_biorxiv_primary_2024_05_14_594150

Other Identifiers

ISSN

2692-8205

E-ISSN

2692-8205

DOI

10.1101/2024.05.14.594150

How to access this item