Fas-mediated neutrophil apoptosis is accelerated by Bid, Bak, and Bax and inhibited by Bcl-2 and Mcl...
Fas-mediated neutrophil apoptosis is accelerated by Bid, Bak, and Bax and inhibited by Bcl-2 and Mcl-1
About this item
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Author / Creator
Croker, Ben A , O'Donnell, Joanne A , Nowell, Cameron J , Metcalf, Donald , Dewson, Grant , Campbell, Kirsteen J , Rogers, Kelly L , Hu, Yifang , Smyth, Gordon K , Zhang, Jian-Guo , White, Michael , Lackovic, Kurt , Cengia, Louise H , O'Reilly, Lorraine A , Bouillet, Philippe , Cory, Suzanne , Strasser, Andreas and Roberts, Andrew W
Publisher
United States: National Academy of Sciences
Journal title
Language
English
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Publication information
Publisher
United States: National Academy of Sciences
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Scope and Contents
Contents
During immune responses, neutrophils must integrate survival and death signals from multiple sources to regulate their lifespan. Signals that activate either the Bcl-2- or death receptor-regulated apoptosis pathways can provide powerful stimuli for neutrophils to undergo cell death, but whether they act cooperatively in parallel or directly cross-t...
Alternative Titles
Full title
Fas-mediated neutrophil apoptosis is accelerated by Bid, Bak, and Bax and inhibited by Bcl-2 and Mcl-1
Authors, Artists and Contributors
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Primary Identifiers
Record Identifier
TN_cdi_crossref_primary_10_1073_pnas_1110358108
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_crossref_primary_10_1073_pnas_1110358108
Other Identifiers
ISSN
0027-8424
E-ISSN
1091-6490
DOI
10.1073/pnas.1110358108
