Hypersensitivity of myelinated A-fibers via toll-like receptor 5 promotes mechanical allodynia in te...
Hypersensitivity of myelinated A-fibers via toll-like receptor 5 promotes mechanical allodynia in tenascin-X-deficient mice associated with Ehlers–Danlos syndrome
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London: Nature Publishing Group UK
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English
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London: Nature Publishing Group UK
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Deficiency of an extracellular matrix glycoprotein tenascin-X (TNX) leads to a human heritable disorder Ehlers–Danlos syndrome, and TNX-deficient patients complain of chronic joint pain, myalgia, paresthesia, and axonal polyneuropathy. We previously reported that TNX-deficient (
Tnxb
−/
−
) mice exhibit mechanical allodynia and hypersen...
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Hypersensitivity of myelinated A-fibers via toll-like receptor 5 promotes mechanical allodynia in tenascin-X-deficient mice associated with Ehlers–Danlos syndrome
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TN_cdi_doaj_primary_oai_doaj_org_article_05ac6b7462934fa48b269f7704dd23bf
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_05ac6b7462934fa48b269f7704dd23bf
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ISSN
2045-2322
E-ISSN
2045-2322
DOI
10.1038/s41598-023-45638-7