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Deficiency of miR-29a/b1 leads to premature aging and dopaminergic neuroprotection in mice

Deficiency of miR-29a/b1 leads to premature aging and dopaminergic neuroprotection in mice

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_09fc3643cd364a36b71bd8715a5f1024

Deficiency of miR-29a/b1 leads to premature aging and dopaminergic neuroprotection in mice

About this item

Full title

Deficiency of miR-29a/b1 leads to premature aging and dopaminergic neuroprotection in mice

Publisher

Lausanne: Frontiers Research Foundation

Journal title

Frontiers in molecular neuroscience, 2022-10, Vol.15, p.978191-978191

Language

English

Formats

Publication information

Publisher

Lausanne: Frontiers Research Foundation

More information

Scope and Contents

Contents

Parkinson’s disease (PD) is a neurodegenerative disorder characterized by progressive degeneration of midbrain dopaminergic neurons. The miR-29s family, including
miR-29a
and
miR-29b1
as well as
miR-29b2
and
miR-29c
, are implicated in aging, metabolism, neuronal survival, and neurological disorders. In this study, the roles...

Alternative Titles

Full title

Deficiency of miR-29a/b1 leads to premature aging and dopaminergic neuroprotection in mice

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_09fc3643cd364a36b71bd8715a5f1024

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_09fc3643cd364a36b71bd8715a5f1024

Other Identifiers

ISSN

1662-5099

E-ISSN

1662-5099

DOI

10.3389/fnmol.2022.978191

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