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TRIM31 promotes Atg5/Atg7-independent autophagy in intestinal cells

TRIM31 promotes Atg5/Atg7-independent autophagy in intestinal cells

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_105465ca7a7148d1ab2b63fadbbbcb8b

TRIM31 promotes Atg5/Atg7-independent autophagy in intestinal cells

About this item

Full title

TRIM31 promotes Atg5/Atg7-independent autophagy in intestinal cells

Publisher

London: Nature Publishing Group UK

Journal title

Nature communications, 2016-05, Vol.7 (1), p.11726-11726, Article 11726

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Autophagy is responsible for the bulk degradation of cytosolic constituents and plays an essential role in the intestinal epithelium by controlling beneficial host–bacterial relationships. Atg5 and Atg7 are thought to be critical for autophagy. However,
Atg5
- or
Atg7
-deficient cells still form autophagosomes and autolysosomes, and are...

Alternative Titles

Full title

TRIM31 promotes Atg5/Atg7-independent autophagy in intestinal cells

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_105465ca7a7148d1ab2b63fadbbbcb8b

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_105465ca7a7148d1ab2b63fadbbbcb8b

Other Identifiers

ISSN

2041-1723

E-ISSN

2041-1723

DOI

10.1038/ncomms11726

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