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NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal m...

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal m...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_156a2a82f24f47d3ae207c2b43a87db2

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

About this item

Full title

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

Publisher

London: Nature Publishing Group UK

Journal title

Scientific reports, 2024-01, Vol.14 (1), p.2326-10, Article 2326

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Hypoxic-ischemic brain damage (HIBD) is a leading cause of neonatal death and neurological dysfunction. Neuroinflammation is identified as one of the crucial pathological mechanisms after HIBD, and natural killer group 2 member D (NKG2D) is reported to be implicated in the pathogenesis of immunoinflammatory diseases. However, the role of NKG2D in n...

Alternative Titles

Full title

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_156a2a82f24f47d3ae207c2b43a87db2

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_156a2a82f24f47d3ae207c2b43a87db2

Other Identifiers

ISSN

2045-2322

E-ISSN

2045-2322

DOI

10.1038/s41598-024-52780-3

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