Glufosinate constrains synchronous and metachronous metastasis by promoting anti‐tumor macrophages
Glufosinate constrains synchronous and metachronous metastasis by promoting anti‐tumor macrophages
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London: Nature Publishing Group UK
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English
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London: Nature Publishing Group UK
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Glutamine synthetase (GS) generates glutamine from glutamate and controls the release of inflammatory mediators. In macrophages, GS activity, driven by IL10, associates to the acquisition of M2‐like functions. Conditional deletion of GS in macrophages inhibits metastasis by boosting the formation of anti‐tumor, M1‐like, tumor‐associated macrophages...
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Full title
Glufosinate constrains synchronous and metachronous metastasis by promoting anti‐tumor macrophages
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TN_cdi_doaj_primary_oai_doaj_org_article_2172c31045fb4eb88d755d43558791aa
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_2172c31045fb4eb88d755d43558791aa
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ISSN
1757-4676,1757-4684
E-ISSN
1757-4684
DOI
10.15252/emmm.201911210
