BNIP3 phosphorylation by JNK1/2 promotes mitophagy via enhancing its stability under hypoxia
BNIP3 phosphorylation by JNK1/2 promotes mitophagy via enhancing its stability under hypoxia
About this item
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Author / Creator
He, Yun-Ling , Li, Jian , Gong, Sheng-Hui , Cheng, Xiang , Zhao, Ming , Cao, Yan , Zhao, Tong , Zhao, Yong-Qi , Fan, Ming , Wu, Hai-Tao , Zhu, Ling-Ling and Wu, Li-Ying
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
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Scope and Contents
Contents
Mitophagy is an important metabolic mechanism that modulates mitochondrial quality and quantity by selectively removing damaged or unwanted mitochondria. BNIP3 (BCL2/adenovirus e1B 19 kDa protein interacting protein 3), a mitochondrial outer membrane protein, is a mitophagy receptor that mediates mitophagy under various stresses, particularly hypox...
Alternative Titles
Full title
BNIP3 phosphorylation by JNK1/2 promotes mitophagy via enhancing its stability under hypoxia
Authors, Artists and Contributors
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Primary Identifiers
Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_24a801bcbed540168f11f3ddd665ae64
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_24a801bcbed540168f11f3ddd665ae64
Other Identifiers
ISSN
2041-4889
E-ISSN
2041-4889
DOI
10.1038/s41419-022-05418-z
