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Phosphorylation of INF2 by AMPK promotes mitochondrial fission and oncogenic function in endometrial...

Phosphorylation of INF2 by AMPK promotes mitochondrial fission and oncogenic function in endometrial...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_2c1e2d9b057f4e2eb2bbf149c8a4b0a4

Phosphorylation of INF2 by AMPK promotes mitochondrial fission and oncogenic function in endometrial cancer

About this item

Full title

Phosphorylation of INF2 by AMPK promotes mitochondrial fission and oncogenic function in endometrial cancer

Publisher

London: Nature Publishing Group UK

Journal title

Cell death & disease, 2024-01, Vol.15 (1), p.65-65, Article 65

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Mitochondria are highly dynamic organelles capable of altering their sizes and shapes to maintain metabolic balance through coordinated fission and fusion processes. In various cancer types, mitochondrial hyperfragmentation has been frequently observed, contributing to the progression of cancer toward metastasis. Inverted formin 2 (INF2), which res...

Alternative Titles

Full title

Phosphorylation of INF2 by AMPK promotes mitochondrial fission and oncogenic function in endometrial cancer

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_2c1e2d9b057f4e2eb2bbf149c8a4b0a4

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_2c1e2d9b057f4e2eb2bbf149c8a4b0a4

Other Identifiers

ISSN

2041-4889

E-ISSN

2041-4889

DOI

10.1038/s41419-024-06431-0

How to access this item