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ER stress inhibition enhances formation of triacylglcerols and protects endothelial cells from lipot...

ER stress inhibition enhances formation of triacylglcerols and protects endothelial cells from lipot...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_44ebdf5ee38d495d9485c5017c5c057d

ER stress inhibition enhances formation of triacylglcerols and protects endothelial cells from lipotoxicity

About this item

Full title

ER stress inhibition enhances formation of triacylglcerols and protects endothelial cells from lipotoxicity

Publisher

England: BioMed Central Ltd

Journal title

Cell communication and signaling, 2024-06, Vol.22 (1), p.304-304, Article 304

Language

English

Formats

Publication information

Publisher

England: BioMed Central Ltd

More information

Scope and Contents

Contents

Elevated concentrations of palmitate in serum of obese individuals can impair endothelial function, contributing to development of cardiovascular disease. Although several molecular mechanisms of palmitate-induced endothelial dysfunction have been proposed, there is no consensus on what signaling event is the initial trigger of detrimental palmitat...

Alternative Titles

Full title

ER stress inhibition enhances formation of triacylglcerols and protects endothelial cells from lipotoxicity

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_44ebdf5ee38d495d9485c5017c5c057d

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_44ebdf5ee38d495d9485c5017c5c057d

Other Identifiers

ISSN

1478-811X

E-ISSN

1478-811X

DOI

10.1186/s12964-024-01682-y

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