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Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndrome

Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndrome

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_4d23ec11c80c4d5eb2ac1d8aff3fa1cc

Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndrome

About this item

Full title

Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndrome

Publisher

Lausanne: Frontiers Research Foundation

Journal title

Frontiers in neuroscience, 2023-09, Vol.17, p.1171797-1171797

Language

English

Formats

Publication information

Publisher

Lausanne: Frontiers Research Foundation

More information

Scope and Contents

Contents

Down syndrome (DS), the most prevalent cause of intellectual disability, stems from a chromosomal anomaly resulting in an entire or partial extra copy of chromosome 21. This leads to intellectual disability and a range of associated symptoms. While there has been considerable research focused on the Ts65Dn mouse model of DS, particularly in the con...

Alternative Titles

Full title

Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndrome

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_4d23ec11c80c4d5eb2ac1d8aff3fa1cc

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_4d23ec11c80c4d5eb2ac1d8aff3fa1cc

Other Identifiers

ISSN

1662-453X,1662-4548

E-ISSN

1662-453X

DOI

10.3389/fnins.2023.1171797

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