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Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amylo...

Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amylo...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_70d51cd3e8b74dccb013aba43110a4f4

Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amyloid-β aggregates

About this item

Full title

Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amyloid-β aggregates

Publisher

England: BioMed Central

Journal title

Journal of neuroinflammation, 2021-06, Vol.18 (1), p.124-124, Article 124

Language

English

Formats

Publication information

Publisher

England: BioMed Central

More information

Scope and Contents

Contents

Alzheimer's disease (AD) and Parkinson's disease (PD) are characterized by brain accumulation of aggregated amyloid-beta (Aβ) and alpha-synuclein (αSYN), respectively. In order to develop effective therapies, it is crucial to understand how the Aβ/αSYN aggregates can be cleared. Compelling data indicate that neuroinflammatory cells, including astro...

Alternative Titles

Full title

Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amyloid-β aggregates

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_70d51cd3e8b74dccb013aba43110a4f4

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_70d51cd3e8b74dccb013aba43110a4f4

Other Identifiers

ISSN

1742-2094

E-ISSN

1742-2094

DOI

10.1186/s12974-021-02158-3

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