AKT controls protein synthesis and oxidative metabolism via combined mTORC1 and FOXO1 signalling to...
AKT controls protein synthesis and oxidative metabolism via combined mTORC1 and FOXO1 signalling to govern muscle physiology
About this item
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Publisher
Germany: John Wiley & Sons, Inc
Journal title
Language
English
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Publisher
Germany: John Wiley & Sons, Inc
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Contents
Background
Skeletomuscular diseases result in significant muscle loss and decreased performance, paralleled by a loss in mitochondrial and oxidative capacity. Insulin and insulin‐like growth factor‐1 (IGF‐1) are two potent anabolic hormones that activate a host of signalling intermediates including the serine/threonine kinase AKT to influence sk...
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Full title
AKT controls protein synthesis and oxidative metabolism via combined mTORC1 and FOXO1 signalling to govern muscle physiology
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TN_cdi_doaj_primary_oai_doaj_org_article_762fe12f3fe3495f9e5c9957980465ca
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_762fe12f3fe3495f9e5c9957980465ca
Other Identifiers
ISSN
2190-5991
E-ISSN
2190-6009
DOI
10.1002/jcsm.12846
