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Hepatitis B virus promotes its own replication by enhancing RAB5A-mediated dual activation of endoso...

Hepatitis B virus promotes its own replication by enhancing RAB5A-mediated dual activation of endoso...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_81d600794b4c48c38d85bfc31e91d737

Hepatitis B virus promotes its own replication by enhancing RAB5A-mediated dual activation of endosomal and autophagic vesicle pathways

About this item

Full title

Hepatitis B virus promotes its own replication by enhancing RAB5A-mediated dual activation of endosomal and autophagic vesicle pathways

Publisher

London: Taylor & Francis Ltd

Journal title

Emerging microbes & infections, 2023-12, Vol.12 (2), p.2261556-2261556

Language

English

Formats

Publication information

Publisher

London: Taylor & Francis Ltd

More information

Scope and Contents

Contents

Chronic hepatitis B virus (HBV) infection remains one of the major global public health concerns, and it develop into liver fibrosis, cirrhosis, and hepatocellular carcinoma. Recent evidence suggests that endosomal and autophagic vesicles are beneficial for HBV replication. However, it has not been well elucidated how HBV exploits such intracellula...

Alternative Titles

Full title

Hepatitis B virus promotes its own replication by enhancing RAB5A-mediated dual activation of endosomal and autophagic vesicle pathways

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_81d600794b4c48c38d85bfc31e91d737

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_81d600794b4c48c38d85bfc31e91d737

Other Identifiers

ISSN

2222-1751

E-ISSN

2222-1751

DOI

10.1080/22221751.2023.2261556

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