Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy
Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy
About this item
Full title
Author / Creator
Souidi, Monia , Resta, Jessica , Dridi, Haikel , Sleiman, Yvonne , Reiken, Steve , Formoso, Karina , Colombani, Sarah , Amédro, Pascal , Meyer, Pierre , Charrabi, Azzouz , Vincenti, Marie , Liu, Yang , Soni, Rajesh Kumar , Lezoualc'h, Frank , Stéphane Blot, D.V.M. , Rivier, François , Cazorla, Olivier , Parini, Angelo , Marks, Andrew R. , Mialet‐Perez, Jeanne , Lacampagne, Alain and Meli, Albano C.
Publisher
Germany: John Wiley & Sons, Inc
Journal title
Language
English
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Publication information
Publisher
Germany: John Wiley & Sons, Inc
Subjects
More information
Scope and Contents
Contents
Background
Duchenne muscular dystrophy (DMD) is an X‐linked disorder characterized by progressive muscle weakness due to the absence of functional dystrophin. DMD patients also develop dilated cardiomyopathy (DCM). We have previously shown that DMD (mdx) mice and a canine DMD model (GRMD) exhibit abnormal intracellular calcium (Ca2+) cycling rel...
Alternative Titles
Full title
Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy
Authors, Artists and Contributors
Author / Creator
Resta, Jessica
Dridi, Haikel
Sleiman, Yvonne
Reiken, Steve
Formoso, Karina
Colombani, Sarah
Amédro, Pascal
Meyer, Pierre
Charrabi, Azzouz
Vincenti, Marie
Liu, Yang
Soni, Rajesh Kumar
Lezoualc'h, Frank
Stéphane Blot, D.V.M.
Rivier, François
Cazorla, Olivier
Parini, Angelo
Marks, Andrew R.
Mialet‐Perez, Jeanne
Lacampagne, Alain
Meli, Albano C.
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Primary Identifiers
Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_86cf946208c7405482fdfb4f8454571b
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_86cf946208c7405482fdfb4f8454571b
Other Identifiers
ISSN
2190-5991
E-ISSN
2190-6009
DOI
10.1002/jcsm.13411