Nitric oxide activates AMPK by modulating PDE3A in human pulmonary artery smooth muscle cells
Nitric oxide activates AMPK by modulating PDE3A in human pulmonary artery smooth muscle cells
About this item
Full title
Author / Creator
Publisher
Oxford: John Wiley & Sons, Inc
Journal title
Language
English
Formats
Publication information
Publisher
Oxford: John Wiley & Sons, Inc
Subjects
More information
Scope and Contents
Contents
Phosphodiesterase 3 (PDE3), of which there are two isoforms, PDE3A and PDE3B, hydrolyzes cAMP and cGMP—cyclic nucleotides important in the regulation of pulmonary vascular tone. PDE3 has been implicated in pulmonary hypertension unresponsive to nitric oxide (NO); however, contributions of the two isoforms are not known. Furthermore, adenosine monop...
Alternative Titles
Full title
Nitric oxide activates AMPK by modulating PDE3A in human pulmonary artery smooth muscle cells
Authors, Artists and Contributors
Author / Creator
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_9debe84f70834594a3b117a0aea6a221
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_9debe84f70834594a3b117a0aea6a221
Other Identifiers
E-ISSN
2051-817X
DOI
10.14814/phy2.14559
