NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9
NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9
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England: BioMed Central Ltd
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Language
English
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Publisher
England: BioMed Central Ltd
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Sustained neuroinflammation strongly contributes to the pathogenesis of pain. The clinical challenge of chronic pain relief led to the identification of molecules such as cytokines, chemokines and more recently matrix metalloproteinases (MMPs) as putative therapeutic targets. Evidence points to a founder member of the matricial CCN family, NOV/CCN3...
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NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9
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TN_cdi_doaj_primary_oai_doaj_org_article_a2fdd8a2b2ec4cbcb4a4c6da29fcfbf4
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_a2fdd8a2b2ec4cbcb4a4c6da29fcfbf4
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ISSN
1742-2094
E-ISSN
1742-2094
DOI
10.1186/1742-2094-9-36
