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Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signa...

Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signa...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_b56819f3c09941b88efc684669d11253

Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signaling pathway

About this item

Full title

Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signaling pathway

Publisher

England: BioMed Central

Journal title

BMC microbiology, 2018-08, Vol.18 (1), p.87-87, Article 87

Language

English

Formats

Publication information

Publisher

England: BioMed Central

More information

Scope and Contents

Contents

The pathogenesis of Helicobacter pylori (H. pylori) infection-induced duodenal ulcer remains to be elucidated. Duodenal mucosal bicarbonate secretion is the most important protective factor against acid-induced mucosal injury. We previously revealed that H. pylori infection downregulated the expression and functional activity of duodenal mucosal cy...

Alternative Titles

Full title

Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signaling pathway

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_b56819f3c09941b88efc684669d11253

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_b56819f3c09941b88efc684669d11253

Other Identifiers

ISSN

1471-2180

E-ISSN

1471-2180

DOI

10.1186/s12866-018-1230-8

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