NOD1/RIP2 signalling enhances the microglia-driven inflammatory response and undergoes crosstalk wit...
NOD1/RIP2 signalling enhances the microglia-driven inflammatory response and undergoes crosstalk with inflammatory cytokines to exacerbate brain damage following intracerebral haemorrhage in mice
About this item
Full title
Author / Creator
Wang, Miao , Ye, Xinchun , Hu, Jinxia , Zhao, Qiuchen , Lv, Bingchen , Ma, Weijing , Wang, Weiwei , Yin, Hanhan , Hao, Qi , Zhou, Chao , Zhang, Tao , Wu, Weifeng , Wang, Yan , Zhou, Mingyue , Zhang, Cong-hui and Cui, Guiyun
Publisher
London: BioMed Central Ltd
Journal title
Language
English
Formats
Publication information
Publisher
London: BioMed Central Ltd
Subjects
More information
Scope and Contents
Contents
Background Secondary brain damage caused by the innate immune response and subsequent proinflammatory factor production is a major factor contributing to the high mortality of intracerebral haemorrhage (ICH). Nucleotide-binding oligomerization domain 1 (NOD1)/receptor-interacting protein 2 (RIP2) signalling has been reported to participate in the i...
Alternative Titles
Full title
NOD1/RIP2 signalling enhances the microglia-driven inflammatory response and undergoes crosstalk with inflammatory cytokines to exacerbate brain damage following intracerebral haemorrhage in mice
Authors, Artists and Contributors
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_d12c0abd62ed42b1b58b73b8415bb45d
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_d12c0abd62ed42b1b58b73b8415bb45d
Other Identifiers
ISSN
1742-2094
E-ISSN
1742-2094
DOI
10.1186/s12974-020-02015-9
