Advanced glycation end-products decreases expression of endothelial nitric oxide synthase through ox...
Advanced glycation end-products decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cells
About this item
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Author / Creator
Ren, Xiaomei , Ren, Liqun , Wei, Qin , Shao, Hua , Chen, Long and Liu, Naifeng
Publisher
England: BioMed Central
Journal title
Language
English
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Publication information
Publisher
England: BioMed Central
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Scope and Contents
Contents
Advanced glycation end-products (AGEs) are elevated under diabetic conditions and associated with insulin resistance, endothelial dysfunction and vascular inflammation in humans. It has been demonstrated that AGEs evoke oxidative and inflammatory reactions in endothelial cells through the interaction with a receptor for AGEs (RAGE). Here, we aimed...
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Full title
Advanced glycation end-products decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cells
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Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_d7f18200c00b4c0e8b05b14098939ef7
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_d7f18200c00b4c0e8b05b14098939ef7
Other Identifiers
ISSN
1475-2840
E-ISSN
1475-2840
DOI
10.1186/s12933-017-0531-9
