AIMP2 accumulation in brain leads to cognitive deficits and blood secretion in Parkinson’s disease
AIMP2 accumulation in brain leads to cognitive deficits and blood secretion in Parkinson’s disease
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England: BioMed Central Ltd
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Language
English
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England: BioMed Central Ltd
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Propagation of neuronal α-synuclein aggregate pathology to the cortex and hippocampus correlates with cognitive impairment in Parkinson's disease (PD) dementia and dementia with Lewy body disease. Previously, we showed accumulation of the parkin substrate aminoacyl-tRNA synthetase interacting multifunctional protein-2 (AIMP2) in the temporal lobe o...
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Full title
AIMP2 accumulation in brain leads to cognitive deficits and blood secretion in Parkinson’s disease
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TN_cdi_doaj_primary_oai_doaj_org_article_e2174386dfb34da982919d1f2bf4c15a
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_e2174386dfb34da982919d1f2bf4c15a
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ISSN
1479-5876
E-ISSN
1479-5876
DOI
10.1186/s12967-024-05666-x
