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Tanshinone IIA suppresses ferroptosis to attenuate renal podocyte injury in diabetic nephropathy thr...

Tanshinone IIA suppresses ferroptosis to attenuate renal podocyte injury in diabetic nephropathy thr...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_e494f3a80b414f0f90f38bfae71af5b3

Tanshinone IIA suppresses ferroptosis to attenuate renal podocyte injury in diabetic nephropathy through the embryonic lethal abnormal visual‐like protein 1 and acyl‐coenzyme A synthetase long‐chain family member 4 signaling pathway

About this item

Full title

Tanshinone IIA suppresses ferroptosis to attenuate renal podocyte injury in diabetic nephropathy through the embryonic lethal abnormal visual‐like protein 1 and acyl‐coenzyme A synthetase long‐chain family member 4 signaling pathway

Publisher

Japan: John Wiley & Sons, Inc

Journal title

Journal of diabetes investigation, 2024-08, Vol.15 (8), p.1003-1016

Language

English

Formats

Publication information

Publisher

Japan: John Wiley & Sons, Inc

More information

Scope and Contents

Contents

Aims/Introduction
Tanshinone IIA (TIIA) is one of the main components of the root of the red‐rooted Salvia miltiorrhiza Bunge. However, the molecular mechanisms underlying TIIA‐mediated protective effects in diabetic nephropathy (DN) are still unclear.
Materials and Methods
High glucose (HG)‐induced mouse podocyte cell line (MPC5) cells we...

Alternative Titles

Full title

Tanshinone IIA suppresses ferroptosis to attenuate renal podocyte injury in diabetic nephropathy through the embryonic lethal abnormal visual‐like protein 1 and acyl‐coenzyme A synthetase long‐chain family member 4 signaling pathway

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_e494f3a80b414f0f90f38bfae71af5b3

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_e494f3a80b414f0f90f38bfae71af5b3

Other Identifiers

ISSN

2040-1116,2040-1124

E-ISSN

2040-1124

DOI

10.1111/jdi.14206

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