miR-200c inhibition and catalase accelerate diabetic wound healing
miR-200c inhibition and catalase accelerate diabetic wound healing
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Author / Creator
D'Agostino, Marco , Sileno, Sara , Lulli, Daniela , De Luca, Naomi , Scarponi, Claudia , Teson, Massimo , Torcinaro, Alessio , De Santa, Francesca , Cirielli, Corrado , Furgiuele, Sergio , Morrell, Chris H , Dellambra, Elena , Odorisio, Teresa , Lakatta, Edward G , Avitabile, Daniele , Capogrossi, M C and Magenta, Alessandra
Publisher
England: BioMed Central Ltd
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Language
English
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Publisher
England: BioMed Central Ltd
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Contents
Reactive oxygen species (ROS) are increased in diabetic conditions and play a causal role in diabetic foot ulcers (DFU). We previously showed that ROS up-regulate miR-200c expression, that in turns causes apoptosis, senescence, ROS upregulation and nitric oxide decrease, leading to endothelial disfunction.
The aim of this study is to dissect miR...
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Full title
miR-200c inhibition and catalase accelerate diabetic wound healing
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TN_cdi_doaj_primary_oai_doaj_org_article_f78b17eaa22f4847ae639cafa6c05f69
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_f78b17eaa22f4847ae639cafa6c05f69
Other Identifiers
ISSN
1021-7770,1423-0127
E-ISSN
1423-0127
DOI
10.1186/s12929-024-01113-7
