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The N6‐methyladenosine modification enhances ferroptosis resistance through inhibiting SLC7A11 mRNA...

The N6‐methyladenosine modification enhances ferroptosis resistance through inhibiting SLC7A11 mRNA...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_f9f46fbe77d44edba01dc7ac8e12e5e1

The N6‐methyladenosine modification enhances ferroptosis resistance through inhibiting SLC7A11 mRNA deadenylation in hepatoblastoma

About this item

Full title

The N6‐methyladenosine modification enhances ferroptosis resistance through inhibiting SLC7A11 mRNA deadenylation in hepatoblastoma

Publisher

United States: John Wiley & Sons, Inc

Journal title

Clinical and Translational Medicine, 2022-05, Vol.12 (5), p.e778-n/a

Language

English

Formats

Publication information

Publisher

United States: John Wiley & Sons, Inc

More information

Scope and Contents

Contents

Background
Solute carrier family 7 member 11 (SLC7A11) is overexpressed in multiple human tumours and functions as a transporter importing cystine for glutathione biosynthesis. It promotes tumour development in part by suppressing ferroptosis, a newly identified form of cell death that plays a pivotal role in the suppression of tumorigenesis. Ho...

Alternative Titles

Full title

The N6‐methyladenosine modification enhances ferroptosis resistance through inhibiting SLC7A11 mRNA deadenylation in hepatoblastoma

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_f9f46fbe77d44edba01dc7ac8e12e5e1

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_f9f46fbe77d44edba01dc7ac8e12e5e1

Other Identifiers

ISSN

2001-1326

E-ISSN

2001-1326

DOI

10.1002/ctm2.778

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