NF-κB RelB suppresses the inflammatory gene expression programs of dendritic cells by competing with...
NF-κB RelB suppresses the inflammatory gene expression programs of dendritic cells by competing with RelA for binding to target gene promoters
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Singapore: Springer Nature Singapore
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English
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Singapore: Springer Nature Singapore
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A group of autoinflammatory disorders termed relopathies arise as a consequence of NF-κB dysregulation. Genetic loss of the NF-κB subunit RelB in humans and mice leads to autoimmunity and lethal multi-organ inflammatory pathology. Our recent study showed that this inflammatory pathology is independent of type I interferon signaling, and further ide...
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NF-κB RelB suppresses the inflammatory gene expression programs of dendritic cells by competing with RelA for binding to target gene promoters
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TN_cdi_doaj_primary_oai_doaj_org_article_fa6b7b6e9d3242289e33fdc2a757f7ce
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_fa6b7b6e9d3242289e33fdc2a757f7ce
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ISSN
2056-5968
E-ISSN
2056-5968
DOI
10.1038/s41421-024-00767-9
