mitoBK.sub.Ca is functionally expressed in murine and human breast cancer cells and potentially cont...
mitoBK.sub.Ca is functionally expressed in murine and human breast cancer cells and potentially contributes to metabolic reprogramming
About this item
Full title
Author / Creator
Bischof, Helmut , Maier, Selina , Koprowski, Piotr , Kulawiak, Bogusz , Burgstaller, Sandra , Jasinska, Joanna , Serafimov, Kristian , Zochowska, Monika , Gross, Dominic , Schroth, Werner , Matt, Lucas , Juarez Lopez, David Arturo , Zhang, Ying , Bonzheim, Irina , Büttner, Florian A , Fend, Falko , Schwab, Matthias , Birkenfeld, Andreas L , Malli, Roland , Lämmerhofer, Michael , Bednarczyk, Piotr , Szewczyk, Adam and Lukowski, Robert
Publisher
eLife Science Publications, Ltd
Journal title
Language
English
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Publication information
Publisher
eLife Science Publications, Ltd
Subjects
More information
Scope and Contents
Contents
Alterations in the function of K.sup.+ channels such as the voltage- and Ca.sup.2+-activated K.sup.+ channel of large conductance (BK.sub.Ca) reportedly promote breast cancer (BC) development and progression. Underlying molecular mechanisms remain, however, elusive. Here, we provide electrophysiological evidence for a BK.sub.Ca splice variant local...
Alternative Titles
Full title
mitoBK.sub.Ca is functionally expressed in murine and human breast cancer cells and potentially contributes to metabolic reprogramming
Authors, Artists and Contributors
Author / Creator
Maier, Selina
Koprowski, Piotr
Kulawiak, Bogusz
Burgstaller, Sandra
Jasinska, Joanna
Serafimov, Kristian
Zochowska, Monika
Gross, Dominic
Schroth, Werner
Matt, Lucas
Juarez Lopez, David Arturo
Zhang, Ying
Bonzheim, Irina
Büttner, Florian A
Fend, Falko
Schwab, Matthias
Birkenfeld, Andreas L
Malli, Roland
Lämmerhofer, Michael
Bednarczyk, Piotr
Szewczyk, Adam
Lukowski, Robert
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_gale_infotracmisc_A795689223
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_gale_infotracmisc_A795689223
Other Identifiers
ISSN
2050-084X
E-ISSN
2050-084X
DOI
10.7554/eLife.92511
