InsP3R–RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle
InsP3R–RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle
About this item
Full title
Author / Creator
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
Journal title
Language
English
Formats
Publication information
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
Subjects
More information
Scope and Contents
Contents
Dysregulated intracellular Ca
2+
handling involving altered Ca
2+
release from intracellular stores via RyR channels underlies both arrhythmias and reduced function in heart failure (HF). Mechanisms linking RyR dysregulation and disease are not fully established. Studies in animals support a role for InsP
3
receptor Ca
2+
ch...
Alternative Titles
Full title
InsP3R–RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle
Authors, Artists and Contributors
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_hal_primary_oai_HAL_hal_04491705v1
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_hal_primary_oai_HAL_hal_04491705v1
Other Identifiers
ISSN
0300-8428
E-ISSN
1435-1803
DOI
10.1007/s00395-022-00967-y
