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PI3Kδ contributes to ER stress-associated asthma through ER-redox disturbances: the involvement of t...

PI3Kδ contributes to ER stress-associated asthma through ER-redox disturbances: the involvement of t...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_nrf_kci_oai_kci_go_kr_ARTI_2701249

PI3Kδ contributes to ER stress-associated asthma through ER-redox disturbances: the involvement of the RIDD–RIG-I–NF-κB axis

About this item

Full title

PI3Kδ contributes to ER stress-associated asthma through ER-redox disturbances: the involvement of the RIDD–RIG-I–NF-κB axis

Publisher

생화학분자생물학회

Journal title

Experimental and Molecular Medicine, 2018, 50(0), , pp.1-12

Language

English

Formats

Publication information

Publisher

생화학분자생물학회

Subjects

Subjects and topics

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Scope and Contents

Contents

Hyperactivation of phosphoinositol 3-kinase (PI3K) has been suggested to be a potential mechanism for endoplasmic reticulum (ER) stress-enhanced airway hyperresponsiveness, and PI3K inhibitors have been examined as asthma therapeutics. However, the regulatory mechanism linking PI3K to ER stress and related pathological signals in asthma have not be...

Alternative Titles

Full title

PI3Kδ contributes to ER stress-associated asthma through ER-redox disturbances: the involvement of the RIDD–RIG-I–NF-κB axis

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Primary Identifiers

Record Identifier

TN_cdi_nrf_kci_oai_kci_go_kr_ARTI_2701249

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_nrf_kci_oai_kci_go_kr_ARTI_2701249

Other Identifiers

ISSN

1226-3613

E-ISSN

2092-6413

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