Intermittent Hypoxia after Transient Focal Ischemia Induces Hippocampal Neurogenesis and c-Fos Expre...
Intermittent Hypoxia after Transient Focal Ischemia Induces Hippocampal Neurogenesis and c-Fos Expression and Reverses Spatial Memory Deficits in Rats
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United States: Public Library of Science
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English
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United States: Public Library of Science
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Memory impairment is a frequent complication of brain ischemia. Neurogenesis is implicated in learning and memory and is regulated by the transcription factor c-Fos. Preconditioning intermittent hypoxia (IH) attenuates ischemia-related memory impairments, but it is not known whether post-ischemia IH intervention has a similar effect. We investigate...
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Intermittent Hypoxia after Transient Focal Ischemia Induces Hippocampal Neurogenesis and c-Fos Expression and Reverses Spatial Memory Deficits in Rats
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TN_cdi_plos_journals_1308335269
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_1308335269
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1932-6203
E-ISSN
1932-6203
DOI
10.1371/journal.pone.0024001