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Role of Prolactin Receptors in Lymphangioleiomyomatosis

Role of Prolactin Receptors in Lymphangioleiomyomatosis

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_1756948147

Role of Prolactin Receptors in Lymphangioleiomyomatosis

About this item

Full title

Role of Prolactin Receptors in Lymphangioleiomyomatosis

Publisher

United States: Public Library of Science

Journal title

PloS one, 2016-01, Vol.11 (1), p.e0146653-e0146653

Language

English

Formats

Publication information

Publisher

United States: Public Library of Science

More information

Scope and Contents

Contents

Pulmonary lymphangioleiomyomatosis (LAM) is a rare lung disease caused by mutations in the tumor suppressor genes encoding Tuberous Sclerosis Complex (TSC) 1 and TSC2. The protein product of the TSC2 gene is a well-known suppressor of the mTOR pathway. Emerging evidence suggests that the pituitary hormone prolactin (Prl) has both endocrine and para...

Alternative Titles

Full title

Role of Prolactin Receptors in Lymphangioleiomyomatosis

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_plos_journals_1756948147

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_1756948147

Other Identifiers

ISSN

1932-6203

E-ISSN

1932-6203

DOI

10.1371/journal.pone.0146653

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