Ammonium tetrathiomolybdate following ischemia/reperfusion injury: Chemistry, pharmacology, and impa...
Ammonium tetrathiomolybdate following ischemia/reperfusion injury: Chemistry, pharmacology, and impact of a new class of sulfide donor in preclinical injury models
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United States: Public Library of Science
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English
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United States: Public Library of Science
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Early revascularization of ischemic organs is key to improving outcomes, yet consequent reperfusion injury may be harmful. Reperfusion injury is largely attributed to excess mitochondrial production of reactive oxygen species (ROS). Sulfide inhibits mitochondria and reduces ROS production. Ammonium tetrathiomolybdate (ATTM), a copper chelator, rele...
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Ammonium tetrathiomolybdate following ischemia/reperfusion injury: Chemistry, pharmacology, and impact of a new class of sulfide donor in preclinical injury models
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TN_cdi_plos_journals_1929384131
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_1929384131
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ISSN
1549-1676,1549-1277
E-ISSN
1549-1676
DOI
10.1371/journal.pmed.1002310
