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Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high...

Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_2279021254

Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high fat diet

About this item

Full title

Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high fat diet

Publisher

United States: Public Library of Science

Journal title

PloS one, 2019-08, Vol.14 (8), p.e0221455-e0221455

Language

English

Formats

Publication information

Publisher

United States: Public Library of Science

More information

Scope and Contents

Contents

Hepatic iron overload, a hallmark of hereditary hemochromatosis, triggers progressive liver disease. There is also increasing evidence for a pathogenic role of iron in non-alcoholic fatty liver disease (NAFLD), which may progress to non-alcoholic steatohepatitis (NASH), fibrosis, cirrhosis and hepatocellular cancer. Mouse models of hereditary hemoc...

Alternative Titles

Full title

Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high fat diet

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_plos_journals_2279021254

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_2279021254

Other Identifiers

ISSN

1932-6203

E-ISSN

1932-6203

DOI

10.1371/journal.pone.0221455

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