Deletion of GSK3β in D2R-expressing neurons reveals distinct roles for β-arrestin signaling in antip...
Deletion of GSK3β in D2R-expressing neurons reveals distinct roles for β-arrestin signaling in antipsychotic and lithium action
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United States: National Academy of Sciences
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Language
English
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United States: National Academy of Sciences
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Several studies in rodent models have shown that glycogen synthase kinase 3 β (GSK3β) plays an important role in the actions of antispychotics and mood stabilizers. Recently it was demonstrated that GSK3β through a β-arrestin2/protein kinase B (PKB or Akt)/protein phosphatase 2A (PP2A) signaling complex regulates dopamine (DA)-and lithium-sensitive...
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Full title
Deletion of GSK3β in D2R-expressing neurons reveals distinct roles for β-arrestin signaling in antipsychotic and lithium action
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TN_cdi_pnas_primary_109_50_20732
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pnas_primary_109_50_20732
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ISSN
0027-8424
E-ISSN
1091-6490
DOI
10.1073/pnas.1215489109
