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Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in gliom...

Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in gliom...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_1891479166

Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in glioma cells

About this item

Full title

Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in glioma cells

Publisher

New York: Nature Publishing Group

Journal title

Oncogenesis (New York, NY), 2017-03, Vol.6 (3)

Language

English

Formats

Publication information

Publisher

New York: Nature Publishing Group

More information

Scope and Contents

Contents

Overexpression of structural maintenance of chromosomes 4 (SMC4) has been reported to be involved in tumor cell growth, migration and invasion, and to be correlated with poor prognosis of cancer patient. However, its clinical signicance and biological role in glioma remain unknown. Herein, we found that SMC4 expression at both mRNA and protein leve...

Alternative Titles

Full title

Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in glioma cells

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_journals_1891479166

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_1891479166

Other Identifiers

E-ISSN

2157-9024

DOI

10.1038/oncsis.2017.8

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