Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in gliom...
Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in glioma cells
About this item
Full title
Author / Creator
Publisher
New York: Nature Publishing Group
Journal title
Language
English
Formats
Publication information
Publisher
New York: Nature Publishing Group
Subjects
More information
Scope and Contents
Contents
Overexpression of structural maintenance of chromosomes 4 (SMC4) has been reported to be involved in tumor cell growth, migration and invasion, and to be correlated with poor prognosis of cancer patient. However, its clinical signicance and biological role in glioma remain unknown. Herein, we found that SMC4 expression at both mRNA and protein leve...
Alternative Titles
Full title
Overexpression of SMC4 activates TGF[beta]/Smad signaling and promotes aggressive phenotype in glioma cells
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_proquest_journals_1891479166
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_1891479166
Other Identifiers
E-ISSN
2157-9024
DOI
10.1038/oncsis.2017.8