Decreased cardiac mitochondrial tetrahydrobiopterin in a rat model of pressure overload
Decreased cardiac mitochondrial tetrahydrobiopterin in a rat model of pressure overload
About this item
Full title
Author / Creator
Publisher
Athens: D.A. Spandidos
Journal title
Language
English
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Publication information
Publisher
Athens: D.A. Spandidos
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Scope and Contents
Contents
Sustained cardiac pressure overload induces mitochondrial dysfunction and apoptosis of cardiomyocytes leading to pathological cardiac hypertrophy and dysfunction. Mitochondrial nitric oxide synthase (NOS) appears to cause uncoupling, which produces reactive oxygen species (ROS) instead of nitric oxide (NO), by a decrease in the cofactor tetrahydrob...
Alternative Titles
Full title
Decreased cardiac mitochondrial tetrahydrobiopterin in a rat model of pressure overload
Authors, Artists and Contributors
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Record Identifier
TN_cdi_proquest_journals_1932350200
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_1932350200
Other Identifiers
ISSN
1107-3756
E-ISSN
1791-244X
DOI
10.3892/ijmm.2013.1236
