Lrfn2-mutant mice display suppressed synaptic plasticity and inhibitory synapse development and abno...
Lrfn2-mutant mice display suppressed synaptic plasticity and inhibitory synapse development and abnormal social communication and startle response
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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English
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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SALM1, also known as LRFN2, is a PSD-95-interacting synaptic adhesion molecule implicated in the regulation of NMDA receptor (NMDAR) clustering largely based on in vitro data, although its in vivo functions remain unclear. Here, we found that mice lacking SALM1/LRFN2 (Lrfn2-/- mice) show a normal density of excitatory synapses but altered excitator...
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Lrfn2-mutant mice display suppressed synaptic plasticity and inhibitory synapse development and abnormal social communication and startle response
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TN_cdi_proquest_journals_2071127150
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2071127150
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E-ISSN
2692-8205
DOI
10.1101/252429
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https://www.proquest.com/docview/2071127150?pq-origsite=primo&accountid=13902
