BAX requires VDAC2 to mediate apoptosis and to limit tumor development
BAX requires VDAC2 to mediate apoptosis and to limit tumor development
About this item
Full title
Author / Creator
Hui San Chin , Van Delft, Mark F , Ninnis, Robert L , Mark, Li X , Tan, Iris Kl , Reljic, Boris , Scicluna, Kristen , Dagley, Laura F , Sandow, Jarrod J , Kelly, Gemma L , Chappaz, Stephane , Khaw, Seong L , Chang, Catherine , Webb, Andrew , Hockings, Colin , Hall, Cathrine , Kueh, Andrew , Ryan, Michael T , Kluck, Ruth M , Bouillet, Philippe , Herold, Marco J , Gray, Daniel Hd , David Cs Huang and Dewson, Grant
Publisher
Cold Spring Harbor: Cold Spring Harbor Laboratory Press
Journal title
Language
English
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Publication information
Publisher
Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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Scope and Contents
Contents
Intrinsic apoptosis is critical for normal physiology including the prevention of tumor formation. BAX and BAK are essential for mediating this process and for the cytotoxic action of many anti-cancer drugs. BAX and BAK are thought to act in a functionally redundant manner and are considered to be regulated similarly. From an unbiased genome-wide C...
Alternative Titles
Full title
BAX requires VDAC2 to mediate apoptosis and to limit tumor development
Authors, Artists and Contributors
Author / Creator
Van Delft, Mark F
Ninnis, Robert L
Mark, Li X
Tan, Iris Kl
Reljic, Boris
Scicluna, Kristen
Dagley, Laura F
Sandow, Jarrod J
Kelly, Gemma L
Chappaz, Stephane
Khaw, Seong L
Chang, Catherine
Webb, Andrew
Hockings, Colin
Hall, Cathrine
Kueh, Andrew
Ryan, Michael T
Kluck, Ruth M
Bouillet, Philippe
Herold, Marco J
Gray, Daniel Hd
David Cs Huang
Dewson, Grant
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_proquest_journals_2071170394
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2071170394
Other Identifiers
E-ISSN
2692-8205
DOI
10.1101/266668
How to access this item
https://www.proquest.com/docview/2071170394?pq-origsite=primo&accountid=13902
