Neuroprotective Effects of AG490 in Neonatal Hypoxic-Ischemic Brain Injury
Neuroprotective Effects of AG490 in Neonatal Hypoxic-Ischemic Brain Injury
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New York: Springer US
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Language
English
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Publisher
New York: Springer US
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Contents
In infants and children, neonatal hypoxic-ischemic (HI) brain injury represents a major cause of chronic neurological morbidity. The transient receptor potential melastatin 2 (TRPM2), a non-selective cation channel that conducts calcium, can mediate neuronal death following HI brain injury. An important endogenous activator of TRPM2 is H
2
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Full title
Neuroprotective Effects of AG490 in Neonatal Hypoxic-Ischemic Brain Injury
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TN_cdi_proquest_journals_2238821036
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2238821036
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ISSN
0893-7648
E-ISSN
1559-1182
DOI
10.1007/s12035-019-01656-z
