EFA6B regulates a stop signal for collective invasion in breast cancer
EFA6B regulates a stop signal for collective invasion in breast cancer
About this item
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Publisher
Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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Language
English
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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Contents
Cancer is initiated by somatic mutations in oncogenes or tumor suppressor genes, however additional mutations provide selective advantages to the tumor cells to resist treatment and develop metastases, therefore identification of secondary mutations is of paramount importance. EFA6B (Exchange Factor for ARF6, B) expression is reduced in breast canc...
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Full title
EFA6B regulates a stop signal for collective invasion in breast cancer
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TN_cdi_proquest_journals_2379939334
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2379939334
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E-ISSN
2692-8205
DOI
10.1101/2020.03.19.998575
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https://www.proquest.com/docview/2379939334?pq-origsite=primo&accountid=13902
