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Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally...

Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2399021204

Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally interact with axons and glia in vivo

About this item

Full title

Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally interact with axons and glia in vivo

Publisher

Cold Spring Harbor: Cold Spring Harbor Laboratory Press

Journal title

bioRxiv, 2020-05

Language

English

Formats

Publication information

Publisher

Cold Spring Harbor: Cold Spring Harbor Laboratory Press

More information

Scope and Contents

Contents

The remyelination failure in multiple sclerosis (MS) is associated with a migration/differentiation block of oligodendroglia. The reason for this block is highly debated. It could result from disease-related extrinsic regulators of the oligodendroglial biology or reflect MS oligodendrocyte intrinsic properties. To avoid confounding immune-mediated...

Alternative Titles

Full title

Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally interact with axons and glia in vivo

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_journals_2399021204

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2399021204

Other Identifiers

E-ISSN

2692-8205

DOI

10.1101/2020.05.05.078642