Excessive fluid shear stress-mediated Klf4 leads to arteriovenous pathogenesis
Excessive fluid shear stress-mediated Klf4 leads to arteriovenous pathogenesis
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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Language
English
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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Background: Vascular networks form, remodel and mature under the influence of both fluid shear stress (FSS) and soluble factors. For example, FSS synergizes with Bone Morphogenic Protein 9 (BMP9) and BMP10 to promote and maintain vascular stability. Mutation of the BMP receptors ALK1, Endoglin or the downstream effector SMAD4 leads to Hereditary He...
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Full title
Excessive fluid shear stress-mediated Klf4 leads to arteriovenous pathogenesis
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TN_cdi_proquest_journals_2718744689
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2718744689
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E-ISSN
2692-8205
DOI
10.1101/2022.07.04.498236
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https://www.proquest.com/docview/2718744689?pq-origsite=primo&accountid=13902
