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1,25-D3 attenuates cerebral ischemia injury by regulating mitochondrial metabolism via the AMPK/AKT/...

1,25-D3 attenuates cerebral ischemia injury by regulating mitochondrial metabolism via the AMPK/AKT/...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2725571146

1,25-D3 attenuates cerebral ischemia injury by regulating mitochondrial metabolism via the AMPK/AKT/GSK3β pathway

About this item

Full title

1,25-D3 attenuates cerebral ischemia injury by regulating mitochondrial metabolism via the AMPK/AKT/GSK3β pathway

Publisher

Lausanne: Frontiers Research Foundation

Journal title

Frontiers in aging neuroscience, 2022-10, Vol.14, p.1015453-1015453

Language

English

Formats

Publication information

Publisher

Lausanne: Frontiers Research Foundation

More information

Scope and Contents

Contents

The brain injury caused by cerebral ischemia-reperfusion is related to mitochondrial damage. Maintaining the normal function of mitochondria, promoting angiogenesis, protecting neuronal cells, and resisting oxidative stress are the keys to functional recovery after acute ischemic stroke. In this study, we established a middle cerebral artery occlus...

Alternative Titles

Full title

1,25-D3 attenuates cerebral ischemia injury by regulating mitochondrial metabolism via the AMPK/AKT/GSK3β pathway

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_journals_2725571146

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2725571146

Other Identifiers

ISSN

1663-4365

E-ISSN

1663-4365

DOI

10.3389/fnagi.2022.1015453

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