Knockdown of TRIM27 alleviated sepsis-induced inflammation, apoptosis, and oxidative stress via supp...
Knockdown of TRIM27 alleviated sepsis-induced inflammation, apoptosis, and oxidative stress via suppressing ubiquitination of PPARγ and reducing NOX4 expression
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Publisher
Cham: Springer International Publishing
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Language
English
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Publisher
Cham: Springer International Publishing
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Contents
Background
Sepsis is a global fatal disease and leads to severe lung injury due to dysfunction of inflammation response. TRIM27 is closely related to the diseased with dysfunction of inflammation response. The aim of this study was to clarify the role and mechanism of TRIM27 in sepsis-induced lung injury.
Methods
The lipopolysaccharide (LP...
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Full title
Knockdown of TRIM27 alleviated sepsis-induced inflammation, apoptosis, and oxidative stress via suppressing ubiquitination of PPARγ and reducing NOX4 expression
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TN_cdi_proquest_journals_2729718336
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2729718336
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ISSN
1023-3830,1420-908X
E-ISSN
1420-908X
DOI
10.1007/s00011-022-01625-8
