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Neonatal hyperoxia in mice triggers long-term cognitive deficits via impairments in cerebrovascular...

Neonatal hyperoxia in mice triggers long-term cognitive deficits via impairments in cerebrovascular...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2746035397

Neonatal hyperoxia in mice triggers long-term cognitive deficits via impairments in cerebrovascular function and neurogenesis

About this item

Full title

Neonatal hyperoxia in mice triggers long-term cognitive deficits via impairments in cerebrovascular function and neurogenesis

Publisher

Ann Arbor: American Society for Clinical Investigation

Journal title

The Journal of clinical investigation, 2022-11, Vol.132 (22), p.1-20

Language

English

Formats

Publication information

Publisher

Ann Arbor: American Society for Clinical Investigation

More information

Scope and Contents

Contents

Preterm birth is the leading cause of death in children under 5 years of age. Premature infants who receive life-saving oxygen therapy often develop bronchopulmonary dysplasia (BPD), a chronic lung disease. Infants with BPD are at a high risk of abnormal neurodevelopment, including motor and cognitive difficulties. While neural progenitor cells (NP...

Alternative Titles

Full title

Neonatal hyperoxia in mice triggers long-term cognitive deficits via impairments in cerebrovascular function and neurogenesis

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_journals_2746035397

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2746035397

Other Identifiers

ISSN

0021-9738

E-ISSN

1558-8238

DOI

10.1172/JCI146095.

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