Insights into the role of glycerophospholipids on the iron export function of SLC40A1 and the molecu...
Insights into the role of glycerophospholipids on the iron export function of SLC40A1 and the molecular mechanisms of ferroportin disease
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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English
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Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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SLC40A1 is the sole iron export protein reported in mammals. In humans, its dysfunction is responsible for ferroportin disease, an inborn error of iron metabolism transmitted as an autosomal dominant trait and observed in different ethnic groups. As a member of the major facilitator superfamily, SLC40A1 requires a series of conformational changes t...
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Insights into the role of glycerophospholipids on the iron export function of SLC40A1 and the molecular mechanisms of ferroportin disease
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TN_cdi_proquest_journals_2922245161
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2922245161
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E-ISSN
2692-8205
DOI
10.1101/2024.02.05.578958
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https://www.proquest.com/docview/2922245161?pq-origsite=primo&accountid=13902
