Phosphoinositide Depletion and Compensatory β-adrenergic Signaling in Angiotensin II-Induced Heart D...
Phosphoinositide Depletion and Compensatory β-adrenergic Signaling in Angiotensin II-Induced Heart Disease: Protection Through PTEN Inhibition
About this item
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Publisher
United States: Cold Spring Harbor Laboratory Press
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Language
English
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Publisher
United States: Cold Spring Harbor Laboratory Press
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Scope and Contents
Contents
Contractile dysfunction, hypertrophy, and cell death during heart failure are linked to altered Ca
handling, and elevated levels of the hormone angiotensin II (AngII), which signals through G
-coupled AT
receptors, initiating hydrolysis of PIP
. Chronic elevation of AngII contributes to cardiac pathology, but the mechanisms linking sust...
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Full title
Phosphoinositide Depletion and Compensatory β-adrenergic Signaling in Angiotensin II-Induced Heart Disease: Protection Through PTEN Inhibition
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TN_cdi_proquest_journals_3172605550
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_3172605550
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ISSN
2692-8205
E-ISSN
2692-8205
DOI
10.1101/2025.02.23.639781
