Reactive oxygen species induce apoptosis in bronchial epithelial BEAS-2B cells by inhibiting the ant...
Reactive oxygen species induce apoptosis in bronchial epithelial BEAS-2B cells by inhibiting the antiglycation glyoxalase I defence: involvement of superoxide anion, hydrogen peroxide and NF-κB
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Boston: Springer US
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English
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Boston: Springer US
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Reactive oxygen species (ROS) are implicated in the regulation of apoptosis through a number of distinct mechanisms depending on cell type and stimulation conditions. Glyoxalase I (GI) metabolizes methylglyoxal (MG) and MG-derived advanced glycation end products (AGEs) known to cause apoptosis. This study examined the possible role of GI among the...
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Full title
Reactive oxygen species induce apoptosis in bronchial epithelial BEAS-2B cells by inhibiting the antiglycation glyoxalase I defence: involvement of superoxide anion, hydrogen peroxide and NF-κB
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TN_cdi_proquest_miscellaneous_1505351116
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1505351116
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ISSN
1360-8185
E-ISSN
1573-675X
DOI
10.1007/s10495-013-0902-y
