Unique role for ATG5 in neutrophil-mediated immunopathology during M. tuberculosis infection
Unique role for ATG5 in neutrophil-mediated immunopathology during M. tuberculosis infection
About this item
Full title
Author / Creator
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
Subjects
More information
Scope and Contents
Contents
Genetic engineering in mice reveals that autophagy is not an essential mechanism in myeloid cells for controlling
Mycobacterium tuberculosis
infection, and that autophagy factor ATG5 protects organisms by regulating neutrophil influx and tissue damage.
Anti-infection role for ATG5 protein
It's widely assumed — based on data from cell cu...
Alternative Titles
Full title
Unique role for ATG5 in neutrophil-mediated immunopathology during M. tuberculosis infection
Authors, Artists and Contributors
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_proquest_miscellaneous_1751989577
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1751989577
Other Identifiers
ISSN
0028-0836
E-ISSN
1476-4687
DOI
10.1038/nature16451
