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Increased Lung Ischemia–Reperfusion Injury in Aquaporin 1–Null Mice Is Mediated via Decreased Hypoxi...

Increased Lung Ischemia–Reperfusion Injury in Aquaporin 1–Null Mice Is Mediated via Decreased Hypoxi...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1808610661

Increased Lung Ischemia–Reperfusion Injury in Aquaporin 1–Null Mice Is Mediated via Decreased Hypoxia-Inducible Factor 2α Stability

About this item

Full title

Increased Lung Ischemia–Reperfusion Injury in Aquaporin 1–Null Mice Is Mediated via Decreased Hypoxia-Inducible Factor 2α Stability

Publisher

United States

Journal title

American journal of respiratory cell and molecular biology, 2016-06, Vol.54 (6), p.882-891

Language

English

Formats

Publication information

Publisher

United States

More information

Scope and Contents

Contents

Aquaporin (AQP) 1, a water channel protein expressed widely in vascular endothelia, has been shown to regulate cell migration, angiogenesis, and organ regeneration. Even though its role in the pathogenesis of lung ischemia-reperfusion (IR) injury has been defined, the functional role of AQP1 during long-term IR resolution remains to be clarified. H...

Alternative Titles

Full title

Increased Lung Ischemia–Reperfusion Injury in Aquaporin 1–Null Mice Is Mediated via Decreased Hypoxia-Inducible Factor 2α Stability

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_1808610661

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1808610661

Other Identifiers

ISSN

1044-1549

E-ISSN

1535-4989

DOI

10.1165/rcmb.2014-0363OC

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