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JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of I...

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of I...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1826679776

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of Intracellular ROS Production

About this item

Full title

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of Intracellular ROS Production

Publisher

New York: Springer US

Journal title

Molecular neurobiology, 2017-07, Vol.54 (5), p.3492-3505

Language

English

Formats

Publication information

Publisher

New York: Springer US

More information

Scope and Contents

Contents

Parthanatos is a form of PARP-1-dependent programmed cell death. The induction of parthanatos is emerging as a new strategy to kill gliomas which are the most common type of primary malignant brain tumor. Oxidative stress is thought to be a critical factor triggering parthanatos, but its underlying mechanism is poorly understood. In this study, we...

Alternative Titles

Full title

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of Intracellular ROS Production

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_1826679776

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1826679776

Other Identifiers

ISSN

0893-7648

E-ISSN

1559-1182

DOI

10.1007/s12035-016-9926-y

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